Originally Posted by Ringman
If you don't mind tell us your favorite ten "positive" mutations.


I don't know that I could pick a favorite but here are some to add to those kindly provided by Idaho Shooter;

1 - Apolipoprotein AI-Milano – removes cholesterol from cell walls
2 - Increased bone density
3- Malaria resistance – a variant of hemoglobin, named HbC provided up to 93% resistance
4- Tetrachromatic vision
Reference for 1-4: https://bigthink.com/daylight-atheism/evolution-is-still-happening-beneficial-mutations-in-humans

Numbers 5 an 6 below are from: https://www.pnas.org/content/113/10/2554#ref-3
as are links to 16 peer-reviewed papers on the very subject you asked for.

5- people who have particular mutations in a gene called SLC30A8 (Solute carrier family 30, member 8) are 65% less likely to get diabetes, even when they have risk factors like obesity (1)

6 - One percent of Northern Europeans are now known to carry a mutation in a gene called CCR-5 that renders a cellular receptor defective and confers total immunity from HIV infection(2)

Flannick J, et al., Go-T2D Consortium, T2D-GENES Consortium
(2014) Loss-of-function mutations in SLC30A8 protect against type 2 diabetes. Nat Genet 46(4):357–363

Liu R, et al.
(1996) Homozygous defect in HIV-1 coreceptor accounts for resistance of some multiply-exposed individuals to HIV-1 infection. Cell 86(3):367–377

Levine ME,
Crimmins EM
(2015) A genetic network associated with stress resistance, longevity, and cancer in humans. J Gerontol Ser A, in press

Freudenberg-Hua Y, et al.
(2014) Disease variants in genomes of 44 centenarians. Mol Genet Genomic Med 2(5):438–450
.
Johnston JJ, et al.
(2015) Individualized iterative phenotyping for genome-wide analysis of loss-of-function mutations. Am J Hum Genet 96(6):913–925

Barzilai N, et al.
(2010) Genetic studies reveal the role of the endocrine and metabolic systems in aging. J Clin Endocrinol Metab 95(10):4493–4500

Schechter CB,
Barzilai N,
Crandall JP,
Atzmon G
(2010) Cholesteryl ester transfer protein (CETP) genotype and reduced CETP levels associated with decreased prevalence of hypertension. Mayo Clin Proc 85(6):522–526

Stitziel NO, et al., Myocardial Infarction Genetics Consortium Investigators
(2014) Inactivating mutations in NPC1L1 and protection from coronary heart disease. N Engl J Med 371(22):2072–2082

Côté M, et al.
(2011) Small molecule inhibitors reveal Niemann-Pick C1 is essential for Ebola virus infection. Nature 477(7364):344–348

Hsu T-H,
Spindler KR
(2012) Identifying host factors that regulate viral infection. PLoS Pathogens 8(7):e1002772

Band G,
Rockett KA,
Spencer CC,
Kwiatkowski DP, Malaria Genomic Epidemiology Network
(2015) A novel locus of resistance to severe malaria in a region of ancient balancing selection. Nature 526(7572):253–257

Mead S, et al.
(2009) A novel protective prion protein variant that colocalizes with kuru exposure. N Engl J Med 361(21):2056–2065
.
Asante EA, et al.
(2015) A naturally occurring variant of the human prion protein completely prevents prion disease. Nature 522(7557):478–481

Iqbal S, et al.
(2015) Serum- glucocorticoid-inducible kinase 1 confers protection in cell-based and in in vivo neurotoxin models via the c-Jun N-terminal kinase signaling pathway. Mol Cell Biol 35(11):1992–2006

Cannon CP, et al., IMPROVE-IT Investigators
(2015) Ezetimibe added to statin therapy after acute coronary syndromes. N Engl J Med 372(25):2387–2397

Friend SH,
Schadt EE
(2014) Translational genomics. Clues from the resilient. Science 344(6187):970–972
Gudbjartsson DF, et al.
(2015) Large-scale whole-genome sequencing of the Icelandic population. Nat Genet 47(5):435–444

Ringman, do you just rely on the logical fallacy of False Dichotomy, thinking that flinging poo at the theory of evolution will somehow support you view? If you have positive evidence for any of your claims please be so kind as to present it.

One unerring mark of the love of the truth is not entertaining any proposition with greater assurance than the proofs it is built upon will warrant. John Locke, 1690